@ 2008.09.10 , 12:44
15

嗑药人生-实验室工艺的6种神奇药

源:popsci。今年看了很多非传统人类,那么让我YY一下,自己也是非人类吧,这篇文章是关于未来药丸的一个集合,看来很像真的,或者你有其他太上老君的金丹,也许可以让我们开开眼。

1.癌症疫苗

抗癌的药物千万种,现如今,战队们的思路是人体接种癌症疫苗。科学家们目前的成果是,在实验室环境改变了人体免疫细胞,然后再送回人体内。如图的胎记瘤疫苗是第一批的实验对象。现在是很复杂,可能以后治疗过程就如接种水痘般简单。

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2.男用避孕药
伟哥的灿烂带来另外的呼声,男用避孕药。研究的方向是,控制精子的生产或者防止射精(ohmygod)。不明朗的未来不仅来自技术的男度,更加是来自男人的欺骗天性,天知道他到底有没有嗑药呢?
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3.抗上瘾药丸
正确的观点是,酒精和毒品上瘾是疾病,而不是以前人们认为的意志力问题。病理学的研究正在进行,鸦片和酒精上瘾是目前比较成功的个案。战队们的野心瞄准了上瘾本身, 神经传导素多巴胺,以及它的习性-它传导快乐,科学家希望将快乐因素剔除出上瘾行为。
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4.吃一片=跑十圈?运动药丸
显然,如图,右边的老鼠咳过药了。加剧新陈代谢来燃烧脂肪,不是一个新的主意,全世界的肥肥们的福音来了,躺着睡觉,就可以瘦下来。
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5.抗衰老的药丸
秦王想要的长生或者还是要找徐福,不过不老或者成为可能。白藜芦醇配合卡路里限制食谱,功效是刺激一个在老鼠和果蝇体内调控衰老的基因。希望也能给人类更多寿元。
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6.聪明丸子
对抗遗忘,帮助体内生成有助于长期记忆的蛋白质,对老年痴呆有效。恐怕,学术奥林匹克也要禁药了。目前的情况是,利他林和其他治疗多动症的药片已经很流行了。当然可以想象,以后期末考前最后一周,一手药丸,一手课本的场景。
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TOTAL COMMENTS: 15+1

  1. 168276

    死磕

  2. 168277

    抗衰老的药,不就是古时候道士们炼的长生不老丹吗?

  3. 168278

    有几张图很有感觉。

  4. 尼古布丁
    @9 years ago
    168284

    4是左边的嗑糖嗑多了吧

  5. dimlau
    @9 years ago
    168285


    资源永远不会平均分配
    就算这些药都是免费发放,偏远地区的人也很难享受到

  6. 夕颜夕颜
    @9 years ago
    168287

    只允许吃一种会吃哪种?……我选抗衰老。。。

  7. 布衣
    @9 years ago
    168288

    最后一个让我想起来机器猫的那个记忆面包片

  8. 168293

    聪明点的都会选聪明药吧 最容易套现的~

  9. 耗子小三
    @9 years ago
    168327

    都是药了…………药药药…………

  10. 阿猫
    @9 years ago
    168355

    会不会有吃抗上瘾药吃到上瘾的?

  11. 那美克星人
    @9 years ago
    168362

    我大学的老板帮WHO做过多年男用避孕药的研究,结果是有无法避免的副作用

    男用避孕还是传说中的埃及避孕法简单高效

  12. 明_kk
    @9 years ago
    168399

    那个运动药丸不会是聪明的发明。 靠加速新陈代谢…… 等你瘦下来你却发现你已经老了10岁了…….

  13. GreyRoar
    @9 years ago
    168487

    没有含笑半步癫?

  14. farOasis
    @9 years ago
    198465

    哪里有售?

  15. 苏姐姐
    @9 years ago
    210113

    各位对4有兴趣的请看这里先………….

    Exercise Pill Is No Replacement For Exercise

    Recently, researchers at the Salk Institute for Biological Studies, a research organization focused on biology and its relation to health, published a study in the journal Cell on the results of a substance that increased exercise endurance without daily exertion when tested in mice. Media reports have described this substance as an “exercise pill,” potentially eliminating the need for exercise. Frank Booth, a University of Missouri expert on the science of inactivity, says the “exercise pill” study did not test all of the commonly known benefits of exercise and taking the pill cannot be considered a replacement for exercise.

    In the Cell paper “Exercise Mimetics” the researchers demonstrated that AMPK-PPARδ pathways, which is a cellular messenger system, can be targeted by orally active drugs to enhance training adaptation or even to increase endurance without exercise. However, Booth cautions that some of the commonly known benefits of exercise were not tested in the Cell paper including:

    – Decreased resting and submaximal exercise heart rate
    – Increased heart stroke volume at all exercise work loads
    – Increased maximal exercise cardiac output
    – Lower blood pressure and arterial stiffness
    – Increased aerobic capacity

    A complete list of the 26 benefits not tested in the paper is included below.

    The prevention of the increased risk of chronic disease produced by lifelong physical inactivity also was not tested in the Cell paper. According to Katzmarzyk & Janssen (Can J Appl Physiol 29:90, 2004), human physical activity decreases the risk of:

    – Coronary artery disease (decreases risk by 45 percent)
    – Stroke (decreases risk by 60 percent)
    – Hypertension (decreases risk by 30 percent)
    – Colon cancer (decreases risk by 41 percent)
    – Breast cancer (decreases risk by 30 percent)
    – Type 2 diabetes (decreases risk by 50 percent)
    – Osteoporosis (decreases risk by 59 percent)

    Until targeting AMPK-PPARδ pathways by drugs is shown to have all the above listed exercise benefits in humans, it is premature to use the term “exercise mimetics” from the very limited observations of the Cell paper, Booth said. Booth’s expectation, based upon his more than 40 years of research experience in exercise and physical inactivity adaptations, is that the drugs in the Cell paper will only partially imitate exercise. In order for any “exercise pill” to counter physical inactivity, the pill must be polygenic, or control many genes at once; therefore the Cell drugs are not likely to provide all of the benefits of comprehensive physical activity. In Booth’s opinion, the drugs used in the Cell paper were not conclusively proven to mimic exercise, contrary to media reports.

    Booth has more than 40 years of research experience in physiological, biochemical, molecular and genetic adaptations that occur during exercise. He is a professor in the MU College of Veterinary Medicine and the MU School of Medicine and a research investigator in the Dalton Cardiovascular Research Center. He is a member of the editorial boards of Journal of Applied Physiology, American Journal of Physiology: Cell Physiology, Physiological Genomics and CardioMetabolic Syndrome.

    Commonly known benefits of exercise not tested in the Cell paper were:

    – Decreased resting and submaximal exercise heart rate
    – Increased heart stroke volume at all exercise work loads
    – Increased maximal exercise cardiac output
    – Lower blood pressure and arterial stiffness
    – Increased aerobic capacity
    – Increased strength and cross-sectional area of skeletal muscle
    – Delayed loss of muscle mass and strength with aging and physical frailty
    – Improved balance and coordination
    – Improved flexibility
    – Reduced osteoporosis
    – Reduced joint stress and back pain
    – Decreased gallstone disease
    – Improved endothelial function
    – Decreased incidence of myocardial ischemia
    – Less myocardial damage from ischemia
    – Decreased oxidative stress
    – Decreased inflammation
    – Improved immune function
    – Decreased liver steatosis and fatty liver disease
    – Improved insulin sensitivity and reduced risk of type 2 diabetes
    – Less likelihood of depression, anxiety, stress and poor psychological well-being
    – Ameliorating hyperlipidemia: lower total cholesterol, higher HDL, and decreased blood triglycerides
    – Improved cognitive function in the elderly
    – Increased blood flow and neurogenesis in the dentate gyrus of the hypothalamus
    – Prevention of the loss of brain volume in the elderly
    – Delay in decline of physiological reserve in organ systems with aging

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